Associate Professor
BIOGRAPHY
Speaker
Abstract
The recent targetable oncogenic driver in NSCLC is MET, a tyrosine kinase receptor expressed mostly in epithelial cells, whose natural ligand is the hepatocyte growth factor (HGF). MET signaling is involved in cell proliferation and cell survival, including the processes of embryonic development, wound healing, and tissue regeneration. With the 1st MET-TKIs approved in FDA and Japan for the treatment of Met Exon 14 skipping mutated NSCLC, the need is arising to profile these patients and understanding of testing methodologies. This talk will cover;
The identification of patients with MET-mutated NSCLC and the importance of testing for MET-mutations
The current unmet needs in MET-mutated NSCLC
Addressing the unmet needs with the emerging targeted therapies for MET-mutated
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